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Despite a wide choice of drugs for stopping the manifestations of the disease, their effectiveness is far from ideal. This is due to the organic nature of the stenosis. It will not be possible to radically affect the bone and cartilage growths that compress the spinal cord with the help of zetia drugs. Therefore, in 75-80% of cases, surgical correction is necessary, since conservative measures fail even to improve the patient's quality of life. Surgical treatment includes the following types of interventions.

How to treat a particular patient is decided jointly by a neurologist and a neurosurgeon. Often, conservative tactics are initially used, and with an insufficient effect or a severe destructive process, an operation is performed. Conservative therapy can be supplemented with folk remedies, but this technique will only slightly alleviate the pain syndrome.

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Since the medicine for organic stenosis does not help enough, conservative therapy can be strengthened by physiotherapy exercises. Gymnastics with a disease helps to increase the range of motion in the spine, stimulates the blood supply to the affected area of the spinal cord and lower extremities. Below are a few typical exercises that are used in combination with medications. Localization in the lumbar region.

Spinal cord compression at the lumbar level is a common variant of the pathology. This is due to the prevalence of degenerative-dystrophic processes of bone and cartilage tissue in this segment. One of the reasons for the development of stenosis in the lumbar region is an increased physical load on the muscular corset, which leads to rapid wear and deformation of the vertebrae. Symptoms of the disease consist of direct irritation of the nervous tissue in the area of the lumbosacral spine, as well as manifestations of compression of the nerve roots. The following signs of the disease are most characteristic.

The main symptom that appears first and worries patients is severe, acute pain in the lumbar region. In the absence of effective therapy, vascular disorders develop, and then motor restrictions in the lower extremities develop. The main task of therapeutic measures is to try to reduce the compression of the spinal cord. Conservative treatment cannot radically solve the problem of compression of the nerve structures, so surgery is the choice. What will help the patient quickly? Consider the main options for operational tactics.

Exercise therapy for spinal stenosis in the lumbar is prescribed before and immediately after surgery. Preliminary preparation includes strengthening the muscular corset through physical training. Exercises for lumbar stenosis are prescribed 3 months before the proposed planned operation. After the intervention, gymnastics during rehabilitation is performed from the second day. Initially, it includes only exercises in bed, and then, as the patient recovers, it changes according to the instructor's decision to more strength.

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The defeat of the cervical spine is the second most common after the pathology in the lumbar segment. If the disease is formed in childhood against the background of a genetic or congenital pathology of bone tissue, then it is considered primary. Secondary stenosis develops on the background of ezetimibe or dorsopathy. A degenerative chronic process is the basis of pathological growths of bone tissue with compression of the spinal cord. Leading symptoms stenosis of the cervical level are as follows.

Both absolute and relative stenosis are dangerous for the patient, as blood circulation in the vertebral arteries is disturbed, which inevitably leads to buy ezetimibe online starvation of the brain. Sagittal stenosis occurs in two variants - it is central and lateral. The first option is characterized by a narrowing of the spinal canal itself, and the second by the pathology of the intervertebral foramen. With lateral stenosis, problems with the blood supply to the brain are most relevant.

Treatment of the disease requires an individual approach and accuracy when choosing a method of surgical correction. This is due to the anatomical fragility of the bone structures, the proximity of the location of the brain. Conservative tactics and exercise therapy often do not give the desired effect, so an operation is used - laminectomy with the installation of a fixing implant. A variant with minidiscectomy is possible, but endoscopic techniques are rarely used. Hypochondroplasia - prerequisites, symptoms (Symptom from Greek - a case, a coincidence, a symptom - one single symptom, a frequent manifestation of any disease, pathological condition or disturbance of any life process), treatment.

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The significant similarity of the symptoms of hypochondroplasia with achondroplasia is justified by the fact that both of these diseases (disturbances in normal life, working capacity) appear as a result of a mutation of the 1st and the same gene - FGFR3, located on chromosome 4, which is responsible for encoding the sequence of fibroblast growth factor-3, which is a transmembrane tyrosine kinase sensor. Normally, it slightly inhibits the growth of fibroblasts and chondrocytes in the growth plates of endochondral bones, which allows it to control the proper development of bone tissue (The structure of tissues of living organisms is studied by the science of histology). With mutations in the FGFR3 gene, fibroblast growth factor-3 is poor, which does not allow it to perform its functions correctly and from time to time leads to the development of thanatoform dysplasia, achondroplasia, or hypochondroplasia.

Hypochondroplasia. prerequisites for the disease (disturbances in normal life, performance), main symptoms (Symptom from Greek - a case, a coincidence, a sign - one single symptom, a frequent manifestation of any disease, pathological condition or disturbance of any life process), treatment and prevention. A hereditary disease that belongs to the group of chondrodysplasias that occur against the background of a violation of the formation of cartilage and certain types of bones, which leads to the development of dwarfism.

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More than 20 mutations of the FGFR3 gene have been identified, resulting in the development of dwarfism by the type of hypochondroplasia. The most common type of deficiency is the deficiency resulting from the substitution of the nitrogenous base at position 1620 of ezetimibe online exon of the gene. As a result, in the protein acquired from such a gene, at position 540, asparaganine is replaced by lysine, which completely changes the tyrosine kinase sensitivity of the sensor. In addition, quite a few other substitutions of nitrogenous bases in the gene and, as a result, amino acids in the acquired protein, which cause the development of hypochondroplasia, have been described. But some information indicates that in patients with a medical picture of this disease (disturbances in normal life, performance), genetic testing did not reveal FGFR3 flaws.